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  • Coagulation factors in the airways in moderate and severe .
    healing, initiated by TF-bearing cells, may propa-gate into exudated bulk plasma, generating exces-sive and physiologically undesirable fibrin that could lead to fibrosis, mucus plug formation, airway narrowing and bronchial hyper-reactivity (BHR).3 6–8 ...
  • Coagulation factors in the airways in moderate and severe .
    physiologically desirable fibrin formation for normal wound healing, initiated by TF-bearing cells, may propagate into exudated bulk plasma, generating excessive and physiologically undesirable fibrin that could lead to fibrosis, mucus plug formation, airway narrowing and
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    Search the history of over 373 billion web pages on the Internet.
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  • Coagulation factors in the airways in moderate and severe .
    healing, initiated by TF-bearing cells, may propa-gate into exudated bulk plasma, generating exces-sive and physiologically undesirable fibrin that could lead to fibrosis, mucus plug formation, airway narrowing and bronchial hyper-reactivity (BHR).3 6–8 ...
  • Coagulation factors in the airways in moderate and severe .
    physiologically desirable fibrin formation for normal wound healing, initiated by TF-bearing cells, may propagate into exudated bulk plasma, generating excessive and physiologically undesirable fibrin that could lead to fibrosis, mucus plug formation, airway narrowing and
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